• Asian-Australasian Journal of Animal Sciences
• /
• v.7 no.3
• /
• pp.357-362
• /
• 1994

Five adult sheep were exposed to intermittent cold for 12 h (18:00-06:00) at an air temperature of $5{\pm}1^{\circ}C$ followed by 12 h (06:00-18:00) at $25{\pm}2^{\circ}C$ over a period of 8 days continuously. Carotid artery blood (Tc), mean skin (Ts) and mean body (Tb = 0.86 Tc + 0.14 Ts) temperatures, heat production rate (HP), respiratory evaporative heat loss, respiration rate (RR) and volume were measured before and after exposure. Tc during the 12 h cold period of intermittent cold exposure was similar to that during the corresponding period in the warm environment, while Tc in the $25^{\circ}C$ of intermittent cold was higher (p < 0.05) than that in the corresponding period in the warm environment. Ts during the cold period markedly decreased (p < 0.001) by about $9^{\circ}C$ when compared with that in the corresponding time period in the warm environment, while Ts during the $25^{\circ}C$ period of intermittent cold recovered to a similar level to that in the warm environment. Tb was lower (p < 0.001) during the cold period of intermittent cold, whereas a slight increase in Tb during the $25^{\circ}C$ period of intermittent cold was significant (p < 0.05) when compared with the value during the similar period in the warm environment. HP was greatly increased (p < 0.001) by cold exposure, followed by an immediate decrease during the first one hour of the 12 h warm period, reaching a similar level to that in the warm environment. A lower (p < 0.05) RR was observed during both the cold and $25^{\circ}C$ period of intermittent cold than during the corresponding periods in the warm environment. The results of the present investigation clearly show that the body temperature of sheep increased during a 12 h warm period following 12 h of exposure to cold. These results suggest that during a warm period of an intermittent cold exposure cycle, heat could be shored in the animal body.

• Asian-Australasian Journal of Animal Sciences
• /
• v.2 no.3
• /
• pp.214-215
• /
• 1989

• Archives of Plastic Surgery
• /
• v.32 no.4
• /
• pp.479-484
• /
• 2005

Raynaud's syndrome causes discolorization, ischemic claudication(pain) and necrosis of the digits through insufficiency in the circulation which is induced by intermittent spasms of the digital arteries. From January, 2002 to December, 2004, 10 patients were surgically treated for Raynaud's syndrome. 9 patients were female and 1 patient was male. 2 patients showed unilateral involvement, 8 patients were operated on both hands. 6 patients had necrotic changes on the finger tips due to the disease. Ages ranged from 21 to 60 with an average of 39.1. Ischemic pain, discolorization, and cold intolerance of the digits were the common symptoms. All patients were evaluated with color doppler before the surgery. Two different procedures were applied according to the severity of the disease: Patients with decreased circulation received, what we call a limited digital sympathectomy, i.e. stripping of the adventitia of the ulnar, radial and common digital arteries. An extended procedure, radical digital sympathectomy, was performed on patients with a complete block of circulation. Stripping of the adventitia in these patients also involved the proper digital arteries. Symptoms like discolorization, ischemic pain, and cold intolerance improved immediately after the surgery. The patients did not suffer from pain even with exposure to cold weather. We conclude that digital sympathectomy could improve the symptoms in Raynaud's patients who do not respond to conservative treatment such as calcium channel blocker and other vasodilators.